Bilateral failure of adduction following orbital decompression.

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Bilateral failure of adduction following orbital decompression.

We report a case of bilateral complete failure of adduction following bilateral translid antralethmoidal orbital decompression. We believe the probable mechanism is neuropraxia (temporary dysfunction) of the third cranial nerves' supply to the medial recti, owing to these nerves' occupying an anatomically abnormal position. Partial recovery of adduction occurred over the ensuing six months.

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Treatment of proptosis in the stable phase of Graves’ orbitopathy differs significantly from that in the acute inflammatory phase. The treatment paradigm employed at the Harkness Eye Institute, reserves surgical decompression in the acute phase for those cases of optic neuropathy that fail to respond to orbital radiotherapy or when radiotherapy is contraindicated. Owing to the 90% success rate ...

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Reduction of Orbital Inflammation following Decompression for Thyroid-Related Orbitopathy

PURPOSE Thyroid-related orbitopathy (TRO) is associated with inflammation, expansion of orbital fat, enlargement of extraocular muscles, and optic neuropathy (ON). We examined the effects of orbital decompression on the inflammatory and congestive signs of TRO in patients who underwent emergent orbital decompression. METHODS This retrospective, consecutive study included patients with ON from...

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ژورنال

عنوان ژورنال: British Journal of Ophthalmology

سال: 1990

ISSN: 0007-1161

DOI: 10.1136/bjo.74.4.239